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Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis

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Last updated 02 junho 2024
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Neutrophil, neutrophil extracellular traps and endothelial cell
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
The distribution of VE-cadherin in cultured endothelial cells
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Lactate released by inflammatory bone marrow neutrophils induces
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Vascular endothelial cell Toll-like receptor pathways in sepsis
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Lactate released by inflammatory bone marrow neutrophils induces
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Vascular permeability in retinopathy is regulated by VEGFR2 Y949
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
PDHA1 hyperacetylation-mediated lactate overproduction promotes
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Endothelial Dysfunction in Atherosclerotic Cardiovascular Diseases
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Frontiers Cardiovascular Dysfunction in COVID-19: Association
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
PDHA1 hyperacetylation-mediated lactate overproduction promotes
Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis
Passing the Vascular Barrier: Endothelial Signaling Processes
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